There may be a way to prevent the worst damage in neurodegenerative diseases like Alzheimer's, Huntington's, ALS, and others.

Understanding more about the interactions that stabilize the phase-shifting FUS protein could reveal new ways to treat ALS and other diseases.

When ALS-affected neurons get more glucose, they live longer and move more efficiently, which could mean more mobility for people with the disease.

After injury or damage, nerve axons can kickstart their own self-destruction. A new gene therapy stops that process in mice.

The interface let users in a clinical trial control an off-the-shelf tablet computer with a virtual mouse they moved with their thoughts.